Why is the difference so dramatic? Who will come out the other side sooner and less damaged?

WutFace can't help it. He / she is ruled by emotion - unable of critical thinking, unable of self-reflection. Expect to be called names... its best to just put him / her on your ignore list.

Oh Mama, tell me where I hurt you.

Case NUMBERS hitting record highs
 

Deaths trending up from post epidemic lows.

https://i.ibb.co/Fndk9QR/744541-E1-3...-A1-E89213.jpg
https://i.ibb.co/Pt26vY0/48-A7524-B-...0960-C2-E2.jpg


so looking at the bright side, at least case mortality is coming down,

Could be:

More young people getting it with lower case mortality
Different strain with higher infectivity but lower case mortality
More community spread and less occupational spread giving Lower infective dose giving lower case mortality
Already exhausted the supply of exceedingly vulnerable so lower case mortality
Timing artifact. Gonna have the case mortality go skyrocketing back to where it was after the inevitable lag.
Other?

Unfortunately I think it is lag. Hospitals are filling now so over the next week to two weeks I expect us to smash through 1k which we are just hiding under heading to 1500. If nothing is done I expect to be 2-3 again in a month.

We're talking lag at work..

Reinfection case was actually a person that donated convalescent antibodies several times. The patient assumed they were immune.

That's all i can say till the case review is published. There are several now being reviewed.

Reinfection or relapse? Some viruses (herpes zoster comes to mind) can be dormant for 50-60 years and then relapse (shingles) as the immunity decreases. Or was it a different strain ?

There is no evidence of dormancy at this time.

Herpes zolster latent phase is much different and wouldn't be expected from covid 19.

If so, we shouldn’t have long to wait to find that out. Yesterday the CDC released the results of a study characterizing follow up information on some of the first COVID fatalities. It was a more in depth study of the comorbidities, racial composition, and clinical course of 10,000 plus COVID fatalities. This is what they found:

So basically, half of those who ultimately were going to die died within 10 days of symptom onset, and were hospitalized only five days before their death occurred.

https://www.cdc.gov/mmwr/volumes/69/...cid=mm6928e1_w

That is far different than the three weeks several posters here have been claiming. Moreover, this is consistent with what was found with the COVID -19s ‘big brother’ SARS back in 2093. Those who died typically had a shorter course of illness - only about five days - than those who survived.

So basically, in five to ten days from the start of those rising numbers, which began about two weeks ago, we should have seen half the effect we were going to see on the deathrate by five days ago. Obviously number of cases continued to go up - but deaths peaked five days ago and are currently plateaued if not starting to fall.

more and more this is looking like a drop in the case-fatality rate, either from younger patients or some other factor.

.
Utter nonsense. How would anyone KNOW about decade long latency in an organism that’s only been studied for six months?


Latent viral infections in humans include herpes simplex, varicella zoster, Epstein-Barr, human cytomegalovirus, adenovirus, and Kaposi's sarcoma. Granted, those are DNA viruses and not RNA viruses, but even Kuru and CJD can exhibit latency, and they aren’t even viruses, just prions. No reason to think it couldn't occur with coronavirus. And certainly no reason to rule it out so soon.

And indeed, researchers are actively considering the possibility:

https://medium.com/microbial-instinc...s-7b305fcfe9a8


https://www.self.com/story/coronavirus-reinfection

And Mouse Hepatitis virus, which IS a coronavirus, has been shown to be capable of going dormant with subsequent reactivating.

https://www.nature.com/articles/nrmi...aft=collection

https://pubmed.ncbi.nlm.nih.gov/4344757/

https://www.researchgate.net/publica...ome_in_AJ_Mice